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Mechanistic aspects of failure of mi...
~
Nalla, Ravi Kiran.
Mechanistic aspects of failure of mineralized tissue.
紀錄類型:
書目-電子資源 : Monograph/item
正題名/作者:
Mechanistic aspects of failure of mineralized tissue.
作者:
Nalla, Ravi Kiran.
面頁冊數:
292 p.
附註:
Chair: Robert O. Ritchie.
附註:
Source: Dissertation Abstracts International, Volume: 65-09, Section: B, page: 4778.
Contained By:
Dissertation Abstracts International65-09B.
標題:
Engineering, Materials Science.
電子資源:
http://pqdd.sinica.edu.tw/twdaoapp/servlet/advanced?query=3146962
ISBN:
0496053493
Mechanistic aspects of failure of mineralized tissue.
Nalla, Ravi Kiran.
Mechanistic aspects of failure of mineralized tissue.
- 292 p.
Chair: Robert O. Ritchie.
Thesis (Ph.D.)--University of California, Berkeley, 2004.
Over the past few decades, there has been increased interest in the structure and properties of mineralized biological tissues like bone and dentin (a structurally simpler analogue of bone that makes up the bulk of the human tooth). In particular, there has been considerable research into the mechanical properties of such tissues and into how they fracture. An understanding of these properties is of great importance from the perspective of developing a realistic framework for life prediction, particularly in light of the effect of microstructural modifications from aging, disease, remodeling, etc. Central to these issues is the resistance to fracture of these materials, and the microstructural mechanisms that are the source of such resistance. Understanding such properties in the context of the inherent hierarchical complexity of the microstructure of these tissues is of obvious importance. However, surprisingly, such questions have largely remained unanswered and to a large extent, even uninvestigated. The present study initially seeks to focus on the fracture and fatigue behavior of dentin and to extend the observations to cortical bone. Accordingly, fracture mechanics based experiments were conducted to evaluate the fracture toughness of dentin in the context of the anisotropy with respect to the underlying microstructure, and discussed in terms of the salient toughening mechanisms active in this material. In light of the presence of such mechanisms, the fracture toughness properties were then evaluated in terms of resistance-curve (R-curve) behavior, i.e., fracture resistance increasing with crack extension. Furthermore, dentin is known to be susceptible to failure under cyclic loading. The "stress-life" and fatigue-crack propagation data obtained through a systematic investigation of the effects of prolonged cyclical loading are discussed. It is concluded that the presence of small incipient flaws in human teeth of the order of 250 mum in size will not radically affect their useful life. The research also extends such observations to microstructurally more complex human cortical bone, and is of importance from the perspective of furthering our understanding of the fracture and fatigue behavior of mineralized tissues and in predicting their failure in vivo.
ISBN: 0496053493Subjects--Topical Terms:
226940
Engineering, Materials Science.
Mechanistic aspects of failure of mineralized tissue.
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Over the past few decades, there has been increased interest in the structure and properties of mineralized biological tissues like bone and dentin (a structurally simpler analogue of bone that makes up the bulk of the human tooth). In particular, there has been considerable research into the mechanical properties of such tissues and into how they fracture. An understanding of these properties is of great importance from the perspective of developing a realistic framework for life prediction, particularly in light of the effect of microstructural modifications from aging, disease, remodeling, etc. Central to these issues is the resistance to fracture of these materials, and the microstructural mechanisms that are the source of such resistance. Understanding such properties in the context of the inherent hierarchical complexity of the microstructure of these tissues is of obvious importance. However, surprisingly, such questions have largely remained unanswered and to a large extent, even uninvestigated. The present study initially seeks to focus on the fracture and fatigue behavior of dentin and to extend the observations to cortical bone. Accordingly, fracture mechanics based experiments were conducted to evaluate the fracture toughness of dentin in the context of the anisotropy with respect to the underlying microstructure, and discussed in terms of the salient toughening mechanisms active in this material. In light of the presence of such mechanisms, the fracture toughness properties were then evaluated in terms of resistance-curve (R-curve) behavior, i.e., fracture resistance increasing with crack extension. Furthermore, dentin is known to be susceptible to failure under cyclic loading. The "stress-life" and fatigue-crack propagation data obtained through a systematic investigation of the effects of prolonged cyclical loading are discussed. It is concluded that the presence of small incipient flaws in human teeth of the order of 250 mum in size will not radically affect their useful life. The research also extends such observations to microstructurally more complex human cortical bone, and is of importance from the perspective of furthering our understanding of the fracture and fatigue behavior of mineralized tissues and in predicting their failure in vivo.
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