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NF-kB-related genetic diseases

Courtois, Gilles.

NF-kB-related genetic diseases

紀錄類型:	書目-電子資源 : Monograph/item
正題名/作者:	NF-kB-related genetic diseasesby Gilles Courtois ... [et al.].
其他作者:	Courtois, Gilles.
出版者:	Cham :Springer International Publishing :2016.
面頁冊數:	xii, 70 p. :ill., digital ;24 cm.
Contained By:	Springer eBooks
標題:	Genetic disorders.
電子資源:	http://dx.doi.org/10.1007/978-3-319-25850-8
ISBN:	9783319258508$q(electronic bk.)

NF-kB-related genetic diseases
NF-kB-related genetic diseases[electronic resource] /by Gilles Courtois ... [et al.]. - Cham :Springer International Publishing :2016. - xii, 70 p. :ill., digital ;24 cm. - SpringerBriefs in biochemistry and molecular biology,2211-9353. - SpringerBriefs in biochemistry and molecular biology..

1) The NF-kB signaling pathways: players and Functions -- 2) Genetic Diseases Affecting the Canonical Pathway of NF-kB Activation -- 3) Genetic Diseases Affecting hte Non-Camonical Pathway of NF-kB Activation -- 4) Genetic Diseases Affecting Ubiquitination Processes in NF-kB Signaling -- 5)Lessons Learned from Studying NF-kB Related Genetic Diseases.

This book presents the diverse clinical, cellular and molecular manifestations of NF-kB-related genetic diseases. It shows that studying patient-related pathologies affecting the components of the NF-kB signaling pathway offers the opportunity to understand the various functions of NF-kB in humans, complementing studies performed with mouse models. In addition, people treating those patients acquire a deeper understanding of the molecular basis of the pathophysiological processes.

ISBN: 9783319258508$q(electronic bk.)

Standard No.: 10.1007/978-3-319-25850-8doiSubjects--Topical Terms:

224987
Genetic disorders.

LC Class. No.: RB155.5

Dewey Class. No.: 616.042

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520 $a This book presents the diverse clinical, cellular and molecular manifestations of NF-kB-related genetic diseases. It shows that studying patient-related pathologies affecting the components of the NF-kB signaling pathway offers the opportunity to understand the various functions of NF-kB in humans, complementing studies performed with mouse models. In addition, people treating those patients acquire a deeper understanding of the molecular basis of the pathophysiological processes.
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